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Recent studies have shown that exosomal crosstalk mechanisms may influence pathways related to immune responses, barrier function, and the gut microbiota (126, 127)

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For instance, it predicts that H 2 S homeostasis will be dysregulated in CBS deficiency-induced hyperhomocysteinemia and suggests that H 2 S-dependent signaling cascades are perturbed in complex diseases like cardiovascular, neurodegenerative, neoplastic, and metabolic diseases where compromised endoplasmic reticulum function is a significant factor

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