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bToxicity specic to the dopamine neuronal population in the cultures was determined by a functional assay that measured the high afnity transport of 3 H-labeled dopamine

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TNF- damages cardiomyocyte mitochondria by inhibiting glycolysis and reducing mitochondrial adenosine triphosphate (ATP) production through promoting oxidative and nitrosative stress, which can result in compromised cardiac systolic function and unfavorable cardiac remodeling [83, 84]

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For me, the impact on recovery time between training sessions has been the most measurable change